Silent Hypoxemia from COVID-19 Explained
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The severe, yet clinically silent, hypoxemia of COVID-19 seen in some patients may be explained by a few basic respiratory pathophysiological principles.
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The severe, yet clinically silent, hypoxemia of COVID-19 seen in some patients may be explained by a few basic respiratory pathophysiological principles.
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PE with syncope is associated with increased risk of short-term mortality, which is explained by the increase in hemodynamic instability seen in these patients as well.
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This is a summary of the major ACR/NKF consensus statements.
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Oral nitroglycerin didn’t seem to be particularly effective for esophageal food impaction in this small, non-blinded single arm study. Only 11.8%, or 2/17 patients, had relief of their symptoms after NTG administration.
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The aortic dissection detection risk score (ADD-RS) is sensitive at a score of ≥1. Adding a D-dimer to the score improves sensitivity even more but at the cost of a slightly lower specificity.
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This small pilot study suggests that vector change defibrillation and double sequential external defibrillation are safe and feasible for termination of refractory ventricular fibrillation and improves the rate of ROSC.
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In asymptomatic patients with INR>10, vitamin K administration was not associated with improved clinical outcomes.
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To improve safety for patients and the health of night shift workers, implement these personal and institutional countermeasures.
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In both an outpatient clinic and ICU population, patients deemed to have low risk penicillin allergies who underwent direct oral challenge of amoxicillin had no reported adverse effects.
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In patients aged 15 to 55 years with benign headaches, low dose IV haloperidol (2.5mg) improved pain in as little as 30 minutes without significant adverse events such as QT prolongation.